Search results for " proteasome inhibition"

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Antiproliferative Properties of a Few Auranofin-Related Gold(I) and Silver(I) Complexes in Leukemia Cells and their Interferences with the Ubiquitin …

2020

A group of triethylphosphine gold(I) and silver(I) complexes, structurally related to auranofin, were prepared and investigated as potential anticancer drug candidates. The antiproliferative properties of these metal compounds were assessed against two leukemia cell lines, i.e., CCRF-CEM and its multidrug-resistant counterpart, CEM/ADR5000. Interestingly, potent cytotoxic effects were disclosed for both series of compounds against leukemia cells, with IC50 values generally falling in the low-micromolar range, the gold derivatives being on the whole more effective than the silver analogues. Some initial structure-function relationships were drawn. Subsequently, the ability of the study compo…

ProteasesProteasome Endopeptidase ComplexAuranofinSilverleukemia cellsPharmaceutical Sciencemetal complexesantiproliferative propertiesArticleAnalytical ChemistryMetallcsh:QD241-44103 medical and health sciencesInhibitory Concentration 500302 clinical medicineGold Compoundslcsh:Organic chemistryCell Line TumorDrug DiscoverymedicineCytotoxic T cellHumansPhysical and Theoretical Chemistry030304 developmental biologyCell Proliferationproteasome inhibition0303 health sciencesLeukemiaChemistryUbiquitinOrganic Chemistryauranofinmedicine.diseaseauranofin metal complexes proteasome inhibition leukemia cells antiproliferative propertiesDrug Resistance MultipleLeukemiaProteasomeBiochemistryChemistry (miscellaneous)Drug Resistance Neoplasm030220 oncology & carcinogenesisvisual_artvisual_art.visual_art_mediumauranofin;metal complexes; proteasome inhibition; leukemia cells; antiproliferative propertiesMolecular MedicineGoldSelectivitymedicine.drugMolecules
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Bortezomib: a new pro-apoptotic agent in cancer treatment.

2010

Bortezomib is a proteasome inhibitor. It targets the ubiquitin-proteasome pathway with subsequent inhibition of the degradation of proteins involved in cell cycle regulation and cancer cell survival. The best known molecular mechanism concerns the inhibition of IkappaB breakdown and the related stabilization of NFkappaB, thus preventing its translocation to the nucleus for the activation of downstream pathways. Bortezomib is the only drug in this class which has been approved for clinical use. It has shown an efficient antitumor effect in a phase III clinical trial (APEX) involving relapsed multiple myeloma patients. Response rate, time to progression and overall survival have been improved…

Cancer ResearchCell cycle checkpointSettore MED/06 - Oncologia MedicaAntineoplastic AgentsApoptosisPharmacologyDexamethasoneBortezomibMiceNeoplasmshemic and lymphatic diseasesAntineoplastic Combined Chemotherapy ProtocolsDrug DiscoverymedicineAnimalsHumansDexamethasoneMultiple myelomaPharmacologyproteasome inhibitionClinical Trials as TopicNeovascularization Pathologicbusiness.industryBortezomibCell CycleNF-kappa Bsolid tumorsmedicine.diseaseBoronic AcidsClinical trialBortezomib; solid tumors; proteasome inhibition.OncologyApoptosisPyrazinesCancer cellProteasome inhibitorCancer researchMultiple MyelomabusinessProteasome InhibitorsBortezomib solid tumors proteasome inhibitionmedicine.drug
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